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Essay: Are Motion Sickness Sensitive People More Sensitive to Smells?

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  • Published: 25 February 2023*
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BACKGROUND: Among the factors contributing to the occurrence of motion sickness (MS)

symptoms, the presence of smells in the environment is very often reported by individuals who are

susceptible to MS. The aim of the present work was to compare olfactory function in MS sensitive

(MS+) and insensitive (MS-) subjects.

METHODS: Olfactory testing included determination of odor detection thresholds, subjective

evaluation of the quality (intensity, hedonicity and familiarity) of three different odorants

(limonene, isovaleric acid and petrol) as well as measures of skin conductance responses to these

three odorants.

RESULTS: Results showed no difference in olfactory sensitivity between MS+ and MS- subjects.

However, findings of both subjective (odor quality self-rating) and objective (psychophysiological

responses) measures did reveal that the affective response to petrol odor was significantly different

in MS+ and in MS- subjects. Indeed, on a scale from 0 (unpleasant) to 10 (pleasant) MS+ subjects

rated petrol odor as more unpleasant (mean = 2.52) than MS- subjects (mean = 4.15) and rise-time

of skin conductance responses to petrol odor was significantly longer in MS+ (mean = 5.98 sec)

compared to MS- subjects (mean =3.22 sec).

DISCUSSION: Our study delves further into the knowledge of the relationship between motion

sickness and olfaction by underlying a modified olfactory perception in motion sickness sensitive

subjects at both psychophysical and psychophysiological levels.

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Introduction

Travel in modern vehicles (cars, boats, planes, helicopters, spacelab, etc.) can cause a large

panel of symptoms such as nausea, headache, and postural discomfort, which are defined as motion

sickness (see 14 for review). Anyone with a healthy vestibular system can become motion sick with

a sufficiently provocative and long motion stimulus. For this reason, a variety of research has been

conducted to get a better understanding of this problem. In addition of being unpleasant, it has been

highlighted that motion sickness can negatively affect performance of complex tasks requiring

sustained performance 20. In particular, motion sickness can even slow air and in-simulators training

for pilots and aircrews 5.

Three main theories offer a clear explanation about motion sickness mechanisms. The ‘toxin

detector’ hypothesis 32 suggests that the brain can identify any mismatch of expected patterns of

vestibular, visual, and kinaesthetic cues as a sign of central nervous system breakdown and a

possible ingested neurotoxin, and thus will initiate vomiting as a defence mechanism. The

vestibular–cardiovascular reflex hypothesis 4 defines motion sickness as a consequence of visceral

discomfort after activation of vestibular autonomic reflexes due to the convergence of vestibular

and autonomic afferent information in the brainstem and cerebellum. The most widely accepted

theory is the sensory conflict or sensory mismatch theory 29, which postulates that motion sickness

originates from a sensory mismatch between actual versus expected invariant patterns of vestibular,

visual and somatosensory inputs. However, whereas motion sickness mechanisms are now well

understood by the scientific community, there is still no actual behavioral or pharmaceutical

technique that could cure motion sickness without side effects.

Among factors that may contribute to motion sickness, such as no view of the road ahead 34, caloric

food 10,22 or nicotine 16, the presence of strong smells is frequently reported by individuals suffering

from motion sickness as a factor that contributes to the occurrence of motion sickness symptoms.

More precisely, some authors suggested that unpleasant odors could contribute to motion sickness

10,13, 34. A recent study investigating motion sickness in rally car co-drivers showed that on-board

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smells were one of the three main risk factors for motion sickness 27. Recently, our team 25

evaluated more precisely the relationship between motion sickness and olfaction. In this study,

subjects were submitted to three sessions of nauseogenic stimulations, Off Vertical Axis Rotation

(OVAR), performed under conditions of olfactory stimulation with limonene (pleasant odor), petrol

(unpleasant odor) or distilled water (as a control). Motion sickness was assessed before, during and

after each OVAR session. This study showed that OVAR consistently increased the induced-motion

sickness. However, the addition of an odor that is pleasant or unpleasant during the rotation did not

affect the occurrence of motion sickness symptoms compared to the control condition. This study

also showed that intensity of odors was significantly increased after OVAR and the intensity was

significantly higher for an unpleasant odor than for a pleasant one. For the hedonicity, OVAR made

unpleasant odor more unpleasant while it made limonene odor slightly more pleasant. Paillard et al.

25 highlighted the lack of influence of odors in motion-induced sickness but an impact of a

nauseogenic test on olfactory perception. Following Paillard et al. 25, one could question whether

sensitivity to odors is higher in motion sickness sensitive subjects.

The aim of the present study was to compare olfactory function in motion sickness sensitive

subjects and in motion sickness insensitive subjects using both psychophysical and

psychophysiological measurement. Psychophysical measures included olfactory detection

thresholds tests and self-rating of intensity, familiarity and hedonicity of three odorants. In addition,

psychophysiological responses to these odorants were analyzed using skin conductance

measurements. Indeed, in the olfactory modality, it is well known that skin conductance can be

modulated by the perception of an odorant 30,35 and specifically that it could be modulated by odor

pleasantness 1,2. Thus, it appears relevant to determine whether psychophysiological responses to

odorants differ between motion sickness sensitive and insensitive subjects.

Methods

Subjects

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The inclusion criteria of the present study were: i) to be non-smokers; ii) to report normal smell

sensitivity and no history of nasal/sinus disease or extensive exposure to chemicals with potential

toxicity; (iii) to be free of any vestibular and neurological disorders.

A group of 85 healthy volunteers (students from the University of Franche-Comté) who fit with

these inclusion criteria were asked to complete the Motion Sickness Susceptibility Questionnaire

(MSSQ12). The first stage of the study was a triage phase where we made sure that our initially

selected subjects has either a MSB score equal or higher than 15 (thus belong to the MS+ group) or

below 2 (thus belong to the MS-group). This criterion has been decided according to Golding15.

Out of these 85 students, 42 volunteers were asked and agreed to take part in the second stage of the

study, i.e. olfactory testing. The sample of subjects included 34 women and 8 men and their age

ranged from 20 to 30 (mean age 22 years 4 months).

The MS+ group, i.e subjects who are very sensitive to motion sickness included 21 subjects (19

women and 2 men, mean age 22 years 5 months; MSB score range between 15 and 40.50). The MSgroup,

i.e subjects who are not sensitive to motion sickness included 21 subjects (15 women and 6

men; mean age 22 years 3 months; MSB score range between 0 and 2).

The study was reviewed and approved by the local ethics committee and declared to the national

authority (N° UF: 1013; DGS 2006/0494) in accordance with the Declaration of Helsinki on

biomedical research involving human subjects. Participation required the completion of an informed

consent form.

For electrodermal recordings 10 subjects in the MS+ group and 12 subjects in the MS- group were

excluded due to low skin conductance responses (SCR <0.02 μS) as described below, or due to the

lack of distinct SCRs during the entire experiment. Thus, 11 subjects in the MS+ group and 9

subjects in the MS- group were available for skin conductance data analyses.

Materials

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Olfactory detection thresholds were determined using n-butanol [C4H10O; molecular weight =

74,12] (Sigma-Aldrich, France). A dilution series (factor 2) was prepared in odorless mineral oil

(Sigma-Aldrich, France). After successive dilutions, the full series include steps 1 to 25 (step 25 is

the highest concentration). Four milliliters of each concentration was placed into glass tubes (7,5 cm

high, 1 cm in diameter at the opening). Another tube was filled with 4 mL of mineral oil only.

For subjective ratings and recording of skin conductance responses three specific odorants were

used: (R)-(+)-limonene [C10H16; molecular weight = 136,23] (Sigma-Aldrich, France), a pleasant

orange-like odor; isovaleric acid [C5H12O2; molecular weight = 102,3] (Sigma-Aldrich, France), an

unpleasant cheesy odor and petrol as a travel related smell. The dilutions used in our study have

been determined according to a pretest carried out on ten subjects. The dilutions that reach a

moderate intensity perception have been chosen for the tests. Specifically, limonene was used

without dilution (100% of the stock solution), while isovaleric acid and petrol were diluted at 50%

in mineral oil and at 25% in water respectively. Nasal stimuli were presented in glass bottles (6 cm

high, 2.5 cm in diameter at the opening) filled with ten milliliters of each solution.

Procedure

The experiment was carried out in two separate sessions. The first session was dedicated to the

psychophysiological evaluation of olfactory function. Olfactory detection thresholds to n-butanol

were determined using a classical ascending binary (stimulus vs. blank) forced-choice method. A

trial consisted in the presentation of two tubes, one being the blank (mineral oil) and the other

containing the dilution of the odorant (n-butanol). The subject indicated which one of the two

randomly presented tubes contained the odorant. Even no sensations were perceived or if no

difference was apparent between the tubes, the subject was required to choose one tube or the other.

No feedback was given regarding the correctness of the responses. Testing began at the weakest

concentration so as to ovoid olfactory receptor's saturation. For each concentration, the test was

performed three

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