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Essay: Reducing Chronic Disease Risk: Getting Healthy with Nutritious Eating and Exercise

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Table of Contents

Introduction:

“Healthy eating lowers the risk of developing chronic medical diseases such as diabetes, hypertension and heart disease”. “Diet is the biggest risk factor”

The key concept to a healthy nutritional lifestyle is consuming a variety of different foods including 5 fruits and vegetables a day, alongside moderate daily exercise. Also considering the intake of potentially harmful foods such as foods which a high in salt and sugar and drinks such as alcohol, minimising the intake of these foods will help you live a healthy life. However individuals who have a high intake of foods rich in salt, sugar and fat, drinking excessive amounts of alcohol have a greater chance of developing chronic diseases which have a direct like to obesity. Diabetes Militias is a disease which is common in individuals who suffer from obesity their lifestyle factors associate closely with the development of the disease.

Diabetes Militias is a metabolic disease which effects the production of insulin in the body. The disorder is quickly becoming prevalent worldwide and casing a worldwide epidemic (Abdulfatai B Olokoba, 2012). The disease can be associated with hyperglycaemia which is very high in patients who suffer with type 2 diabetes (A.Ramachandran, 2014), hyperglycaemia is the medical term for high glucose levels( NHS England 2012) this indicates that body is unable to produce insulin or unable to use insulin in the correct method.

Diabetes can be recognised as a heterogeneous and complex disease which effects people across the population at many different stages of life (Sobha Sivaprased, 2017). Type 1 diabetes refers to a genetic disease, whereas the onset for type 2 diabetes is obesity and unhealthy lifestyles, currently type 2 diabetes affects more than 2.6 million people and is on the rise (Bassett, 2005)Patients who suffer with type 2 diabetes will encounter macrovascular and microvascular complications which will lead to physical and psychological distress (Sudesna Chatterjee, 2017) hence there is a link between type 2 diabetes and depression (Square, 2016).

Type 2 diabetes which is also known as non-insulin depended develops due to the body becoming unable to respond to the action of insulin which is produced by the pancreas {add reference here} and is illustrated as metabolic syndrome (Abdulfatai B Olokoba, 2012). Currently studies have shown that around 366 million people have Diabetes Mellitus, with around 80% having type 2 (Abdulfatai B Olokoba, 2012). This indicates that diabetes is a worldwide medical issue which needs to be resolved as the incidence of diabetes will carry on increasing. The cause of type 2 diabetes links primary to lifestyle factors (Abdulfatai B Olokoba, 2012), one of the treatments include to eat a healthier more balanced diet which includes 5 fruits and vegetables as well as 2 litres of water which equates to around 11 cups and carry out daily moderate exercise.

Also to prevent the patient from experiencing symptoms of type 2 diabetes she has been provided with a new diet which is high in carbohydrates and low in fat intake, this will improve her health so that she can control the diabetes, as there is currently no cure for diabetes. You can manage the symptoms which will control the blood glucose level, patients would be advised to consume high fibre food, whole grains and fruit and vegetables, told to monitor blood sugar as well as regular exercise. Diet plays a role in the management of type 2 diabetes as a healthier diet and lifestyle will prevent the development of type 2 diabetes (L, 2009). A diet which is high in fat will increase calorie intake, taking in more calories will increase weight which after a period of time will cause the development of obesity. Body mass causes the onset of insulin resistance which is the cells not responding to insulin, thus the pancreas increases the production of insulin so that glucose can enter cells (Abdullah S Al- Goblan, 2014). Therefore it is vital that a healthy diet which is high in fibre, fruits and vegetables is emphasized in patients with diabetes militias.

The aim of the case study is to understand the effects diet has on type 2 diabetes and what can be done to help improve lifestyle for someone who suffers with the disease. The importance of the study is to understand the role a healthy diet plays in preventing the onset of vigorous life threating diseases.

Method:  

A 3 day plan was recorded for a 40 year old women which included her food intake for 3 meals that were breakfast, lunch and dinner. Breakfast was eaten at 8am for the 3 days, lunch was eaten at 12pm and diner was eaten at 7pm, she also consumed 2 bottles of 500 ml water and on the third day she drank 2 cans of beer and 20 ml spirit. Diet plan 6 was used to analyse her dietary components from the software a dietary assay was produced.

Her 3 day meal record was provided for (see appendix A) which was then placed into the diet plan 6 software where her dietary components were generated and results were used to create her assay. Using the eat well plate and the diet plan software an ideal healthier diet has been recommended for the women who suffers type 2 diabetes to ensure her energy requirements were meet for a women of her age and suffering with her condition.

Fig1: The software programme layout is shown, the patients data was entered (height and weight) this is key as this information will be used to generate the patients nutrients and energy intakes from the foods that she consumes over the course of the 3 days. Once this information has been entered, the next stage would be entering in the patient’s food intake. Also so that you can access the analysis at any time to modify and adjust it is vital that you input your registration number so that the data can be found when needed.  

Fig 2: An example of how the meals were inserted onto the software can be seen by figure 2. You can see that there were 3 meals breakfast, lunch and dinner. This was carried out for the course of the 3 days and the amount of each food which was consumed has been added too. This information had then generated graphs and tables which will be very important when analysing the patient’s nutrient intake and suggesting a healthier diet for her.

Results:

Table 1: Table showing analysis of selected nutrients for original diet

The table shows the analysis of nutrients from the patient’s original diet and RDA was shown for some nutrients as well. The patient had a high energy intake of 9660 which per day was 3220 and per meal was 1073, her fat intake was high (416.2). Also the table shows that comparing her nutrient intake with the recommend daily allowance the patient had been getting a higher intake than she actually needed ie for potassium the RDA is 2000 whereas the women was in taking 5363 per day which shows her intake was well above the daily recommended allowance.

Table 1- Nutrient analysis for original diet

Nutrient

Unit

Intake

Per day

RDA

Per meal

Edible proportion

0.98 m

0.98

0.98

Water

g

60.96.0

2032.0

677.3

Total nitrogen

g

96.92 e

32.31

10.77

Protein

g

589.8

196.6

65.5

Fat

g

416.2

138.7

46.2

Available Carbohydrate (mse)

g

886.5

295.5

98.5

Energy

kcal

9660

3220

1073

Energy

kJ

40479

13493

4498

Starch(mse)

g

710.8 m

236.9

79.0

Total sugars(mse)

g

175.3 m

58.4

19.5

Non-milk extrinsic sugars

g

18.7 m

6.2

2.1

Glucose

g

15.6 m

5.2

1.7

Fructose

g

22.7 m

7.6

2.5

Sucrose(mse)

g

103.9 m

34.6

11.5

Maltose (mse)

g

10.8 m

34.6

1.2

Lactose (mse)

g

6.2 m

2.1

0.7

Non-starch polysaccharides

g

28.9 m

9.6

3.2

Total dietary fibre ( AOAC method)

g

37.7 m

12.6

4.2

Saturated fatty acids

g

123.8 m

41.3

13.8

Mono-unsaturated fatty acids

g

146.2 m

48.7

16.2

Poly-unsaturated fatty acids

g

74.3

24.8

8.3

Total trans fatty acids

g

12.54

4.18

1.39

Cholesterol

mg

2941.9 m

980.6

326.9

Sodium (Na)

mg

7780 m

2593

864

Potassium (K)

mg

16088 m

5363

2000

1788

Calcium (Ca)

mg

2376 m

792

800

264

Magnesium (Mg)

mg

1626 m

542

375

181

Phosphorus (P)

mg

7708 m

2569

700

856

Iron (Fe)

mg

59.90 m

19.97

14.00

6.66

Copper (Cu)

mg

6.12 m

2.04

1.00

0.68

Zinc (Zn)

mg

57.11 m

19.04

10.00

6.35

Chloride (Cl)

mg

10808 m

3603

800

1201

Manganese (Mn)

mg

13.26 m

4.42

2.00

1.47

Selenium (Se)

ug

374.5 m

124.8

55.0

41.6

Iodine (I)

ug

680.9

227.0

150.0

75.7

Retinol

ug

1630 m

543

181

Carotene

ug

18084 m

6028

2009

Vitamin D

ug

43.76 m

14.59

5.00

4.86

Vitamin E

mg

46.44 m

15.8

12.00

5.16

Thiamin

mg

7.11 m

2.37

1.10

0.79

Riboflavin

mg

6.51 m

2.17

1.40

0.72

Niacin

mg

168.16 m

56.05

16.00

18.68

Tryptophan divided by 60

mg

110.254 m

36.751

12.250

Vitamin B6

mg

10.44 m

3.48

1.40

1.16

Vitamin B12

ug

47.9 m

16.0

2.5

5.3

Total Folate

ug

788 m

263

200

88

Pantothenic acid

mg

26.58 m

8.86

6.00

2.95

Biotin

ug

258.4 m

86.1

50.0

28.7

Vitamin C

mg

151 m

50

80

17

Key:

E- Value estimated

M- Missing value

Table 2: Showing analysis of selected nutrients for new diet

The table shows the analysis of nutrients for the suggested diet and RDA was shown for some key nutrients. The energy intake for the new diet is 1393 which was for the course of 3 meals. The fat intake for this diet is 38.2 and the carbohydrate intake was 187.0 suggesting that the new diet is better than her previous one. Also from the RDA it shows that the nutrient intakes were within the recommend daily allowance or they weren’t very high for example for potassium the RDA is 2000 and the intake for the new diet was 2048.

Table 2- Nutrient analysis for suggested/ new diet

Nutrient

Unit

Intake

Per day

RDA

Per meal

Edible proportion

0.98

0.98

0.98

Water

g

1288.0 m

1288.0

429.3

Total nitrogen

g

13.76 e

13.76

429.3

Protein

g

85.2

85.2

28.4

Fat

g

38.2

38.2

12.7

Available Carbohydrate (mse)

g

187.0 e

187.0

62.3

Energy

kcal

1393

1393

464

Energy

kJ

5867

5867

1956

Starch(mse)

g

81.5 m

81.5

27.2

Total sugars(mse)

g

41.9 m

41.9

14.0

Non-milk extrinsic sugars

g

13.9 m

13.9

4.6

Glucose

g

9.8 m

9.8

3.3

Fructose

g

11.7 m

11.7

3.9

Sucrose(mse)

g

19.3 m

19.3

6.4

Maltose (mse)

g

Lactose (mse)

g

0.7 m

0.7

0.2

Non-starch polysaccharides

g

9.3 m

9.3

3.1

Total dietary fibre ( AOAC method)

g

12.4

12.4

4.1

Saturated fatty acids

g

4.6 m

4.6

1.5

Mono-unsaturated fatty acids

g

6.9 m

6.9

2.3

Poly-unsaturated fatty acids

g

8.2 m

8.2

2.7

Total trans fatty acids

g

0.08 m

0.08

0.03

Cholesterol

mg

139.1 m

139.1

46.4

Sodium (Na)

mg

1912 m

1912

637

Potassium (K)

mg

2048

2048

2000

683181

Calcium (Ca)

mg

181 m

181

800

60

Magnesium (Mg)

mg

230 m

230

375

77

Phosphorus (P)

mg

1251 m

1251

700

417

Iron (Fe)

mg

7.47 m

7.47

14.00

2.49

Copper (Cu)

mg

0.67 m

0.67

1.00

0.22

Zinc (Zn)

mg

5.70 m

5.70

10.00

1.90

Chloride (Cl)

mg

2592 m

2592

800

864

Manganese (Mn)

mg

4.18m

4.18

2.00

1.39

Selenium (Se)

ug

79.6 m

79.6

55.0

26.5

Iodine (I)

ug

47.4 m

47.4

150.0

15.8

Retinol

ug

52 m

52

17

Carotene

ug

751 m

751

250

Vitamin D

ug

7.62 m

7.61

5.00

2.54

Vitamin E

mg

7.99m

7.99

12.00

2.66

Thiamin

mg

0.91 m

0.91

1.10

0.30

Riboflavin

mg

0.72 m

0.72

1.40

0.24

Niacin

mg

29.63 m

29.63

16.00

9.88

Tryptophan divided by 60

mg

14.565m

14.565

4.855

Vitamin B6

mg

1.53 m

1.53

1.40

0.51

Vitamin B12

ug

4.2 m

4.2

2.5

1.4

Total Folate

ug

129 m

129

200

43

Pantothenic acid

mg

3.49

3.49

6.00

1.16

Biotin

ug

25.6

25.6

50.0

8.5

Vitamin C

mg

86 m

86

80

29

Key:

E- Value estimated

M- Missing value

Table 3: Table showing source energy from original diet

The table shows the sources of energy from the original diet. Most of the calories had come from the fat that was consumed in the patient’s diet as the calories from fat were 3746 and this is where she had gained most of her energy from as fat gave her an energy source of 1249 per day which equates to 38%.

Calories from

Total

Per day

Per 100g

Percent (%)

UK Food (%)

UK Total (%)

Protein

2359

786

28

24.4

15

15

Carbohydrate

3324

1108

39

34.4

50

47

Fat

3746

1249

44

38.8

35

33

Alcohol

146

49

2

1.5

5

Fibre

75m

25

1

0.8

Total

9651m

3217

115

100.0

100%

100%

Table 4: Table showing energy source from suggested diet

The table shows the sources of energy from the suggested/ new diet. Most of the calories would come from the carbohydrates that would be consumed, the calories from the carbohydrates is 701 whereas fat is only 344, she would be gaining most of her energy from the carbohydrates which will be 234 which would equate to 49.7 of energy gained from carbohydrates compared to the rest of the nutrients.

Calories from

Total

Per day

Per 100g

Percent (%)

UK Food (%)

UK Total (%)

Protein

341

114

20

24.1

15

15

Carbohydrate

701

234

41

49.7

50

47

Fat

344

115

20

24.4

35

33

Alcohol

0

0

0

0.0

5

Fibre

25

8

1

1.8

Total

1411

470

83

100.0

100%

100%

Graph 1: Graph showing nutrient intake as percentage of RDA

Fig 3: The graph shows the amount of nutrients the patient was consuming as a percentage of RDA. From the graph it shows that chloride had the highest consumption of nutrients per RDA which was 450% and calcium which was the lowest at 99%.

Fig 4: The graph is a continuation from figure 3(graph 1). Vitamin C had the lowest intake which was 63% and vitamin B12 had the highest which was 639%.

Graph 2: Pie chart showing nutrient contribution to energy for her original diet

Fig 5: The pie chart shows the nutrients that gave the patient the most amount of energy where starch at 27.6%, protein and 24.4% and saturated fat at 11.5%. Thus showing the patient got most of her energy from starch.

Graph 3: Pie chart showing nutrient contribution to energy for her new/suggested diet

Fig 6: The pie chart shows the nutrients that gave the most energy were starch and protein, protein at 24.1% and starch at 21.1%, most of the energy had come from the protein which would be consumed.

Graph 4: Bar chart showing the sources of enegry intake from nutreints conusmed per day and per 100 grams for original diet  

Fig 7: The bar chart shows that the patient had gained most of her energy from fats and the least from fibres, she had not consumed any fibres in her diet for the 3 days.

Graph 5: Bar chart showing the sources of enegry intake from nutreints conusmed per day and per 100 grams for suggested/new diet

Fig 8: The bar chart shows that the patient would gain most of her energy from carbohydrates and the least from alcohol as no alcohol would be consumed.

Figure 9: Calculation to show total energy expenditure by patient and extra energy intake:

Energy expenditure via daily activity = 1200 calories/day

Womens BMR = 65.6 + (9.6 x weight in kg) + (1.7 x height in centimetre) – (4.7 x age in years)

   = 65.6 + (9.6 x 109) + (1.7 x 172) – (4.7 x 40)

  = 65.6 + 1046.4 + 292.4 – 188

  = 1216.4

Total energy intake per day = 3217 kcalories/ day (gained from table 3)

10% of total intake  = 3217 – 10% = 321.7 k calories/day

Total energy expenditure by patient = energy expenditure via daily actvivity + BMR + 10% of total intake  

  = 1200 + 1216.4 + 321.7

   = 2738.1 kcalories

Extra energy intake of patient = Total energy intake per day – total energy expenditure by patient

   = 3217 kcalories – 2738.1 kcalories

   = 478.9 calories

Discussion:

From the dietary analysis carried out on my diet plan software the original diet that the patient was consuming very high amounts of fats and proteins she had a nutrient intake on fat of 416.2 and protein intake of 589.9 (see table 1) which suggests that her diet was the main cause of the development type 2 diabetes this is supported by (Julie A, 2002) who conducted an animal study and found there is a link between impaired insulin action and high fat diets (Julie A, 2002). This is also supported by (Kinsell, 1959) who were one of the first to find a connection between fat consumption and insulin action. Therefore the new diet/ suggested diet will include less fats and protein and more carbohydrates this will control the patients symptoms of diabetes, as there is no cure patients are advised to eat healthier. This change can be seen in table 2 were the carbohydrate intake was the greatest at 187.0 whereas fat intake is 38.2. Also table 1 shows that for most nutrient the patient was consuming more than the recommended daily allowance (RDA) is, this can be toxic can cause damage to the organs for example from table 1 the patient had a high intake of potassium which was 5363 when the RDA was only 2000, therefore this can lead to hyperkalaemia which is high levels of potassium in the blood, this is a life threating disease (Lehnhardt, 2011).

Moreover as the diet is high in fat and protein implies that the energy will generally be high as protein and fats are high sources of energy, table 3 shows that the patient had an intake of 3217kcal of energy however the daily intake for women is 2000, therefore too much energy can lead to feelings of anxiety, palpitations and high blood pressure which can lead to heart attacks and heart disease as she has too much energy. Therefore the fat will build up in her blood the substances which are found in blood from fatty foods are triglycerides and cholesterols, if you have high amounts of these lipids in your blood it can cause heart attacks, thus the more fat you eat the greater the chances are for you to develop a heart attack. From graphs 4 and 5 you can clearly see her energy intakes and what food causes her to have higher energy intakes, thus from graph 5 you can see that the change in her diet has also changed her energy intakes as the fat ha reduced.  Consequently it is very important for someone who already has type 2 diabetes and is obese to change or control their diet so that they do not develop high blood lipids thus the new diet for the patient had a daily energy intake of 470 per day. Comparing the patient’s energy percentages to the UK Food % you can see that her percentages for protein and fat were higher than then UK food percentages (see table 3). From table 4 you can see that the percentages were similar or lower than the UK food percentages thus showing that the new/ suggested diet will help improve the patients help and control her symptoms.

Graph one shows the RDA intakes for the nutrients that she was eating, we can see that her intake for vitamin B12 was very high, she had a percentage of 639% which shows that comparing her intake to the RDA it is too high and not needed, therefore showing that her original diet has been giving her a high intake of nutrients that she didn’t need or that aren’t as important as others, for example vitamin C is more important than V B12, yet her intake was only 63% for vitamin C thus implying that the nutrients that her body needed more off she was not getting, therefore when creating her new diet plan this factor was taken into consideration ( see appendix 1). Graph 2 shows that the patient was had high intakes of starch but she was also eating foods that would cause her symptoms to become worse for example she had consumed alcohol and saturated fats and mono unsaturated fats thus causing her blood sugar levels to increase therefore the inulin in her body will become impaired as the patient is continuing to eat food which is harmful to her. Graph 3 shows that the patient had an intake of all the key nutrients she needed including fibres which she lacked from her original diet and her intake of sugars and fats were greatly reduced and she had no consumption of alcohol.

Figure 9 shows that the patient had an extra calorie intake of 478.9 calories and for someone with type 2 diabetes this can be very harmful as extra food intakes leads to fat built up which in turn causes the onset of diseases such as heart disease and can lead to heart failure as the blood lipids will become blocked and if the blood cannot move around the body freely it will lead to blood clots which lead to heart attacks. In conclusion it is very important to control fat intake and reduce the intake if you have to especially for someone such as the patient in the case study who suffers type 2 diabetes.

References

A.Ramachandran. (2014). Know the signs and symtopms of diabetes . Indian journal of medical research , 579-581.

Abdulfatai B Olokoba, O. A. (2012). Type 2 Diabetes Mellitus: A Review of Current Trends. Oman medical journal , 269-273.

Abdullah S Al- Goblan, M. A.-A. (2014). Mechanism linking diabetes mellitus and obesity. Diabetes, metabolic syndrome and obesity , 587-591.

Bassett, M. T. (2005). Diabetes is Epidemic. Amercian public health assocation , 1496.

IDF. (2018, October 25). International diabetes atlas. Retrieved from IDF: http://www.idf.org/diabetesatlas/5e/Update2012

Julie A, M. a. (2002). Dietary Fat and the Development of Type 2 Diabetes. diabetes care journal , 620-622.

Kinsell. (1959). Dietary fats and the diabetic patient. New england journal of medicine, 431-434.

L, W. (2009). Understanding the role of diet in type 2 diabetes prevention. British journal of community nursing , 374-379.

Lehnhardt, A. (2011). Pathogenesis, diagnosis and management of hyperkalemia. Pediatric nephrology , 377-384.

Sobha Sivaprased, A. t. (2017). Diabetes; a dynamic disease . The lancet, 2163.

Square, A. L. (2016). The association between Diabetes mellitus and Depression. Journal of medicine and life , 120-125.

Sudesna Chatterjee, K. M. (2017). Type 2 dabetes. The lancet, 2239-2251.

England, N. (2017). Diabetes. [online] NHS England. Available at: https://www.england.nhs.uk/diabetes/ [Accessed 20 Oct. 2018

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